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The clostridia : molecular biology and pathogenesis Edited by Julian Rood ... [et al.] Published by Academic Press (1997) |
Book description
The clostridia are a group of bacteria of considerable medical and economic importance and
include species responsible for generating the most potent toxins known to humans.
The Clostridia: Molecular Biology and Pathogenesis is a unique work, comprising the
most complete reference on the clostridia for over 20 years, bringing together the results from
some of the most innovative and exciting research in the past decade. Using a principle-oriented
rather than taxonomic approach, the results from molecular biology research are placed in the
context of their clinical significance, and the disease process as a whole.
This state-of-the-art work is truly comprehensive, covering and integrating the diverse topics of
genetics, physiology, pathogenesis and cell biology.
Written and edited by world-renowned authorities, material is presented to give the reader an
up-to-date knowledge of the pathogenic species of this important genus. Background
information is followed by details of the genetics, molecular biology, biochemistry and disease
mechanisms. The structure, function and mode of action of toxins and other virulence
determinants is clearly presented. As such, this work will prove essential for students, teachers,
research microbiologists, infectious disease clinicians, toxin specialists, and all those working in
medical or veterinary bacteriology, microbial genetics and the pharmaceutical industries.
Key Features
* Covers appropriate medical and veterinary topics
* Contains authoritative contributions by international experts
* Presents the current state of knowledge and areas for future research
* Truly comprehensive--covers topics from molecular biology and physiology
About the Author
Dr. Julian Rood is a Senior Lecturer in the Department of Microbiology at Monash University.
Borrow it: Monash University Library members can borrow this book. View the catalogue record for details.
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